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CAST Chinese Acute Stroke Trial ; Collaborative Group. CAST: randomized placebo-controlled trial of early aspirin use in 20.000 patients with acute ischaemic stroke. Lancet 1997; 349: 1641-1649. CAVATAS Investigators. Endovascular versus surgical treatment in patients with carotid stenosis in the Carotid and Vertebral Artery Transluminal Angioplasty Study CAVATAS ; : a randomized trial. Lancet 2001; 357: 1729-1737. Chan F, Ching J, Hung L, et al. Clopidogrel versus aspirin and esomeprazole to prevent recurrent ulcer bleeding. N Engl J Med 2005; 352 3 ; : 238-44. Chimowitz M, Kokkinos J, Strong J, et al. The warfarin-aspirin symptomatic intracranial disease study. Neurology 1995; 45: 1488-1493. Chimowitz MI, Lynn MJ, Howlett-Smith H, et al. Comparison of warfarin and aspirin for symptomatic intracranial arterial stenosis. N Engl J Med 2005; 352 13 ; : 1305-16. Colhoun HM, Betteridge DJ, Durrington PN, et al. Primary prevention of cardiovascular disease with atorvastatin in type 2 diabetes in the Collaborative Atorvastatin Diabetes Study CARDS ; : multicentre randomised placebo-controlled trial. Lancet 2004; 364: 685-696. Collaborative Group of the Primary Prevention Project PPP ; . Low-dose aspirin and vitamin E in people at cardiovascular risk: a randomised trial in general practice. Lancet 2001; 357: 89-95. Collins R, Peto R, MacMahon S, et al. Blood pressure, stroke and coronary heart disease. Part 2, short- term reductions in blood pressure: overview of randomized drug trials in their epidemiological context. Lancet 1990; 335: 827-838. Daffertshofer M, Grips E, Dempfle C, et al. Heparin during acute ischemic stroke. Present data and clinical situation. Nervenarzt 2003; 74: 307319. Dahlof B, Devereux RB, Kjeldsen SE, et al. Cardiovascular morbidity and mortality in the losartan intervention for endpoint reduction in hypertension study LIFE ; : a randomised trial against atenolol. Lancet 2002; 359: 995-1003. Department of Health and Human Services, Food and Drug Administration. Internal analgesic, antipyretic, and antirheumatic drug products for over-the counter human use. Final rule for professional labeling of aspirin, buffered aspirin and aspirin in combination with antacid drug products. Federal register 1998; 63: 56802-56819. Diener H, Bogousslavsky J, Brass L, et al. Acetylsalicylic acid on a background of clopidogrel in high-risk patients randomised after recent ischaemic stroke or transient ischaemic attack: The MATCH trial and estradiol!

4. Conclusions The catalytic behavior of the Fe3 + Fe2 + system in the electro-Fenton degradation of chlorophene solutions with 0.05 M Na2SO 4 and different Fe3 + concentrations of pH 3.0 mainly depends on the cathode tested. The cells with either a Pt or BDD anode and an O2diffusion cathode yield a large accumulation of electrogenerated H2O 2 while Fe 3 + content remains practically constant. Chlorophene falls more rapidly with raising Fe3 + concentration up to 8.0 mM, since more amount of oxidant OH is formed from Fenton's reaction 1 ; due to the higher amount of Fe2 + regenerated at the O 2-diffusion cathode from reaction 10 ; . In contrast, the latter reaction is so fast at a carbon-felt cathode that Fe2 + is largely accumulated, but H2O 2 is electrogenerated in small extent. This is feasible by the much slower oxidation of Fe 2 and BDD from reaction 10 ; , as explained by the pseudo-first-order rate constants determined. In these systems the antimicrobial decay is enhanced with raising current thanks to the higher generation of H2O2 and Fe2 + leading to greater amount of OH from Fenton's reaction 1 ; , only being required 0.2 mM Fe3 + to obtain its maximum production under all applied currents. The removal rate of chlorophene is always lower in the cells with BDD than with Pt, because Fe2 + is less accumulated since it is also oxidized with peroxodisulfate generated at the BDD anode. A second-order rate constant of 1.000.01 ; x1010 M-1 s-1 is determined for the reaction between chlorophene and OH in solution from the method of competitive kinetics with benzoic acid. Concentrated solutions of the antimicrobial are poorly decontaminated in the Pt O2 diffusion cell with 4.0 mM Fe3 + , whereas total mineralization is achieved using the BDD O 2 diffusion cell with 4.0 mM Fe 3 high current and the Pt carbon felt and BDD carbon felt cells with 0.2 mM Fe3 + . The initial chlorine is completely released as chloride ion, which remains stable in solution using a Pt anode, but it is oxidized to Cl2 on BDD. At the early stages of treatment, the efficiency for the degradation process in the cells increases in the order: Pt O 2 diffusion BDD O 2 diffusion BDD carbon felt Pt carbon felt, although it always rises with decreasing current. The hard oxidation of final Fe3 + -oxalate complexes and other undetected products with OH in the medium and at the Pt surface accounts for the poor degradation in the Pt O2 diffusion cell. These species are completely mineralized at a BDD anode at high current due to the great production of reactive OH on its and famotidine.
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